PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32787917-0	2020	Exosomes derived from atorvastatin-pretreated MSC accelerate diabetic wound repair by enhancing angiogenesis via AKT/eNOS pathway.	Atorvastatin	22-34	nitric oxide synthase 3	Rattus norvegicus	117-121	
32787917-8	2020	The role of miRNAs and AKT/eNOS signaling pathway in the promoted angiogenesis of ATV-Exos were assessed with their inhibitors.	Atorvastatin	82-85	nitric oxide synthase 3	Rattus norvegicus	27-31	
32787917-12	2020	And AKT/eNOS pathway was activated by ATV-Exos and the pro-angiogenic effects of ATV-Exo were attenuated after the pathway being blocked.	Atorvastatin	38-41	nitric oxide synthase 3	Rattus norvegicus	8-12	
32787917-12	2020	And AKT/eNOS pathway was activated by ATV-Exos and the pro-angiogenic effects of ATV-Exo were attenuated after the pathway being blocked.	Atorvastatin	81-84	nitric oxide synthase 3	Rattus norvegicus	8-12	
32787917-14	2020	CONCLUSIONS: Exosomes originated from ATV-pretreated MSCs might serve as a potential strategy for the treatment of diabetic skin defects through enhancing the biological function of endothelial cells via AKT/eNOS pathway by upregulating the miR-221-3p.	Atorvastatin	38-41	nitric oxide synthase 3	Rattus norvegicus	208-212