PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32750045-10	2020	THS-induced over-production of interleukin-6 (IL-6) and tumor necrosis factor-alpha (TNF-alpha) in BALF and serum was suppressed by MaR1.	THYMIDINE-5'-(DITHIO)PHOSPHATE	0-3	interleukin 6	Rattus norvegicus	31-44	
32750045-10	2020	THS-induced over-production of interleukin-6 (IL-6) and tumor necrosis factor-alpha (TNF-alpha) in BALF and serum was suppressed by MaR1.	THYMIDINE-5'-(DITHIO)PHOSPHATE	0-3	interleukin 6	Rattus norvegicus	46-50	
32750045-10	2020	THS-induced over-production of interleukin-6 (IL-6) and tumor necrosis factor-alpha (TNF-alpha) in BALF and serum was suppressed by MaR1.	THYMIDINE-5'-(DITHIO)PHOSPHATE	0-3	tumor necrosis factor	Rattus norvegicus	56-83	
32750045-10	2020	THS-induced over-production of interleukin-6 (IL-6) and tumor necrosis factor-alpha (TNF-alpha) in BALF and serum was suppressed by MaR1.	THYMIDINE-5'-(DITHIO)PHOSPHATE	0-3	tumor necrosis factor	Rattus norvegicus	85-94	
32750045-11	2020	Besides, the TLR4/p38MAPK/NF-kappaB pathway activation in THS-induced rats were inhibited by MaR1 treatment.	THYMIDINE-5'-(DITHIO)PHOSPHATE	58-61	toll-like receptor 4	Rattus norvegicus	13-17	
32750045-12	2020	CONCLUSIONS Our study showed that MaR1 could effectively alleviated THS-induced lung injury via inhibiting the excitation of the TLR4/p38MAPK/NF-kappaB pathway in THS-induced rats, suggesting that MaR1 might be a novel agent for lung damage treatment.	THYMIDINE-5'-(DITHIO)PHOSPHATE	68-71	toll-like receptor 4	Rattus norvegicus	129-133	
32750045-12	2020	CONCLUSIONS Our study showed that MaR1 could effectively alleviated THS-induced lung injury via inhibiting the excitation of the TLR4/p38MAPK/NF-kappaB pathway in THS-induced rats, suggesting that MaR1 might be a novel agent for lung damage treatment.	THYMIDINE-5'-(DITHIO)PHOSPHATE	163-166	toll-like receptor 4	Rattus norvegicus	129-133