PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32571750-1	2021	Acetaminophen (APAP) hepatotoxicity is mediated by N-acetyl-p-benzoquinone imine (NAPQI), a highly toxic metabolite generated by cytochrome P450 2E1 (CYP2E1).	Acetaminophen	0-13	cytochrome P450, family 2, subfamily e, polypeptide 1	Mus musculus	129-148	
32571750-1	2021	Acetaminophen (APAP) hepatotoxicity is mediated by N-acetyl-p-benzoquinone imine (NAPQI), a highly toxic metabolite generated by cytochrome P450 2E1 (CYP2E1).	Acetaminophen	0-13	cytochrome P450, family 2, subfamily e, polypeptide 1	Mus musculus	150-156	
32571750-1	2021	Acetaminophen (APAP) hepatotoxicity is mediated by N-acetyl-p-benzoquinone imine (NAPQI), a highly toxic metabolite generated by cytochrome P450 2E1 (CYP2E1).	Acetaminophen	15-19	cytochrome P450, family 2, subfamily e, polypeptide 1	Mus musculus	129-148	
32571750-1	2021	Acetaminophen (APAP) hepatotoxicity is mediated by N-acetyl-p-benzoquinone imine (NAPQI), a highly toxic metabolite generated by cytochrome P450 2E1 (CYP2E1).	Acetaminophen	15-19	cytochrome P450, family 2, subfamily e, polypeptide 1	Mus musculus	150-156	
32571750-2	2021	Thus, pathological conditions increasing CYP2E1 activity can favour APAP-induced liver injury, which is characterized by massive hepatocellular necrosis and secondary sterile inflammation.	Acetaminophen	68-72	cytochrome P450, family 2, subfamily e, polypeptide 1	Mus musculus	41-47