PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32335272-9	2020	Remarkably, we showed that administration of an antisense oligonucleotide (ASO) inhibitor of miR-129-5p to an ALS animal model, SOD1 (G93A) mice, result in a significant increase in survival and improved the neuromuscular phenotype in treated mice.	Oligonucleotides	58-73	microRNA 1295a	Homo sapiens	93-103	
32335272-9	2020	Remarkably, we showed that administration of an antisense oligonucleotide (ASO) inhibitor of miR-129-5p to an ALS animal model, SOD1 (G93A) mice, result in a significant increase in survival and improved the neuromuscular phenotype in treated mice.	Oligonucleotides	58-73	superoxide dismutase 1, soluble	Mus musculus	128-132	
32335272-9	2020	Remarkably, we showed that administration of an antisense oligonucleotide (ASO) inhibitor of miR-129-5p to an ALS animal model, SOD1 (G93A) mice, result in a significant increase in survival and improved the neuromuscular phenotype in treated mice.	Oligonucleotides, Antisense	75-78	microRNA 1295a	Homo sapiens	93-103	
32335272-9	2020	Remarkably, we showed that administration of an antisense oligonucleotide (ASO) inhibitor of miR-129-5p to an ALS animal model, SOD1 (G93A) mice, result in a significant increase in survival and improved the neuromuscular phenotype in treated mice.	Oligonucleotides, Antisense	75-78	superoxide dismutase 1, soluble	Mus musculus	128-132