PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32195121-2	2020	N2O prolonged and recurrent administration is known to affect vitamin B12 metabolism with subsequent clinical consequences.	Nitrous Oxide	0-3	NADH:ubiquinone oxidoreductase subunit B3	Homo sapiens	70-73	
32195121-3	2020	We report herein the case of a 13-year-old girl with sickle cell disease exhibiting severe neurological and biochemical signs of functional vitamin B12 deficiency due to prolonged and repeated exposure to N2O.	Nitrous Oxide	205-208	NADH:ubiquinone oxidoreductase subunit B3	Homo sapiens	148-151	
32195121-4	2020	This was an incentive to prospectively investigate functional vitamin B12 deficiency in patients affected by sickle cell disease regularly exposed to N2O.	Nitrous Oxide	150-153	NADH:ubiquinone oxidoreductase subunit B3	Homo sapiens	70-73	
32195121-7	2020	The clinical case highlight the potential severe deleterious effects of N2O over exposure on B12 vitamin metabolism in particular in patients affected with sickle cell disease.	Nitrous Oxide	72-75	NADH:ubiquinone oxidoreductase subunit B3	Homo sapiens	93-96