PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31917285-0	2020	Icariside II attenuates eosinophils-induced airway inflammation and remodeling via inactivation of NF-kappaB and STAT3 in an asthma mouse model.	baohuoside I	0-12	signal transducer and activator of transcription 3	Mus musculus	113-118	
31917285-10	2020	In both OVA-induced mouse model of asthma and TGF-beta1-induced ASMCs, Icariside II decreased IkappaBalpha degradation, nuclear translocation of NF-kappaB p65 and STAT3 phophorylation, indicating an inactivation of NF-kappaB and STAT3 in the presence of Icariside II.	baohuoside I	71-83	signal transducer and activator of transcription 3	Mus musculus	163-168	
31917285-10	2020	In both OVA-induced mouse model of asthma and TGF-beta1-induced ASMCs, Icariside II decreased IkappaBalpha degradation, nuclear translocation of NF-kappaB p65 and STAT3 phophorylation, indicating an inactivation of NF-kappaB and STAT3 in the presence of Icariside II.	baohuoside I	71-83	signal transducer and activator of transcription 3	Mus musculus	229-234	
31917285-11	2020	Therefore, we demonstrate that Icariside II attenuates eosinophils-induced airway inflammation and remodeling in asthmatic mice and inhibits TGF-beta1-induced cell proliferation and migration in ASMCs via suppressing NF-kappaB and STAT3 signalings.	baohuoside I	31-43	signal transducer and activator of transcription 3	Mus musculus	231-236