PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31306741-6	2019	In vitro, corticosterone increased GR and HDAC7 whereas reduced the H3K9ac level of KLF4 and KLF4/Nephrin expression.	Corticosterone	10-24	nuclear receptor subfamily 3, group C, member 1	Rattus norvegicus	35-37	
31306741-7	2019	KLF4 over-expression reversed the reduction of Nephrin expression, knockdown of HDAC7 and GR antagonist RU486 partially reversed the inhibitory effects of corticosterone on H3K9ac level and KLF4 expression.	Corticosterone	155-169	nuclear receptor subfamily 3, group C, member 1	Rattus norvegicus	90-92	
31306741-8	2019	In conclusion, PCE caused podocyte developmental toxicity in male offspring, which was associated with corticosterone-induced low-functional programming of KLF4 through GR/HDAC7/H3K9ac pathway.	Corticosterone	103-117	nuclear receptor subfamily 3, group C, member 1	Rattus norvegicus	169-171