PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30679163-0	2019	LIMS1 Promotes Pancreatic Cancer Cell Survival under Oxygen-Glucose Deprivation Conditions by Enhancing HIF1A Protein Translation.	Oxygen	53-59	LIM zinc finger domain containing 1	Homo sapiens	0-5	
30679163-3	2019	We determined the mechanistic role of LIM and senescent cell antigen-like-containing domain protein 1 (LIMS1) in cancer cell survival under oxygen-glucose deprivation conditions.	Oxygen	140-146	LIM zinc finger domain containing 1	Homo sapiens	38-101	
30679163-3	2019	We determined the mechanistic role of LIM and senescent cell antigen-like-containing domain protein 1 (LIMS1) in cancer cell survival under oxygen-glucose deprivation conditions.	Oxygen	140-146	LIM zinc finger domain containing 1	Homo sapiens	103-108	
30679163-9	2019	Increased LIMS1 expression was pivotal for tumor cells to survive in the oxygen-glucose deprivation conditions.	Oxygen	73-79	LIM zinc finger domain containing 1	Homo sapiens	10-15	
30679163-11	2019	Furthermore, LIMS1 promoted HIF1A protein translation by activating AKT/mTOR signaling, while hypoxia-inducible factor 1 (HIF1) transactivated LIMS1 transcription, thus forming a positive feedback loop in PDAC cell adaptation to oxygen deprivation stress.	Oxygen	229-235	LIM zinc finger domain containing 1	Homo sapiens	13-18	
30679163-13	2019	CONCLUSIONS: LIMS1 promotes pancreatic cancer cell survival under oxygen-glucose deprivation conditions by activating AKT/mTOR signaling and enhancing HIF1A protein translation.	Oxygen	66-72	LIM zinc finger domain containing 1	Homo sapiens	13-18	
30679163-14	2019	LIMS1 is crucial for tumor adaptation to oxygen-glucose deprivation conditions and is a promising therapeutic target for cancer treatment.	Oxygen	41-47	LIM zinc finger domain containing 1	Homo sapiens	0-5