PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30598523-2	2019	Here, we found T-acute lymphoblastic leukemia (T-ALL) cells acquire resistance to dexamethasone (DEX)-mediated killing through abnormal activation of Akt, resulting in inhibition of the FoxO3a/Bim pathway.	Dexamethasone	82-95	AKT serine/threonine kinase 1	Homo sapiens	150-153	
30598523-2	2019	Here, we found T-acute lymphoblastic leukemia (T-ALL) cells acquire resistance to dexamethasone (DEX)-mediated killing through abnormal activation of Akt, resulting in inhibition of the FoxO3a/Bim pathway.	Dexamethasone	97-100	AKT serine/threonine kinase 1	Homo sapiens	150-153	
30598523-5	2019	Inhibition of Akt is most effective at restoring sensitivity to DEX of GC-resistant lymphocytes in vitro and in vivo, but shows significant hepatotoxicity in vivo.	Dexamethasone	64-67	AKT serine/threonine kinase 1	Homo sapiens	14-17