PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30542713-5	2019	As high glucose is able to induce the expression of epidermal growth factor (EGF), which is intimately related with tumor progression, the aim of this study was to evaluate whether curcumin could influence the high glucose-induced EGF/EGFR pathway and the biological activity of pancreatic cancer cells.	Curcumin	181-189	epidermal growth factor receptor	Homo sapiens	235-239	
30542713-13	2019	In addition, EGF-modulated activation of EGFR, ERK and Akt, as well as the expression of uPA and E-cadherin were inhibited by curcumin.	Curcumin	126-134	epidermal growth factor receptor	Homo sapiens	41-45	
30542713-14	2019	Taken together, the present study indicates that curcumin suppresses hyperglycemia-driven EGF-induced invasion and migration of pancreatic cancer cells by inhibiting the EGF/EGFR signaling pathway and its downstream signaling molecules including ERK and Akt.	Curcumin	49-57	epidermal growth factor receptor	Homo sapiens	174-178