PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30362049-7	2019	NaB treatment increased the formation of autolysosome and expression of phosphorylated liver kinase B1 (LKB1), AMP-activated protein kinase (AMPK), and acetyl-CoA carboxylase (ACC).	nab	0-3	protein kinase AMP-activated catalytic subunit alpha 1	Homo sapiens	111-139	
30362049-7	2019	NaB treatment increased the formation of autolysosome and expression of phosphorylated liver kinase B1 (LKB1), AMP-activated protein kinase (AMPK), and acetyl-CoA carboxylase (ACC).	nab	0-3	protein kinase AMP-activated catalytic subunit alpha 1	Homo sapiens	141-145	
30362049-8	2019	Treatment with compound C (an inhibitor of AMPK) and siRNA-mediated knockdown of AMPK and LKB1 significantly attenuated NaB-induced autophagy in CRC cells.	nab	120-123	protein kinase AMP-activated catalytic subunit alpha 1	Homo sapiens	81-85	
30362049-9	2019	Collectively, these findings indicated that LKB1 and AMPK are critical for NaB-mediated autophagy and may act as the novel targets for colorectal cancer therapy in the future.	nab	75-78	protein kinase AMP-activated catalytic subunit alpha 1	Homo sapiens	53-57