PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
3033417-0	1987	The benzodiazepine agonist clonazepam potentiates the effects of gamma-aminobutyric acid on alpha-MSH release from neurointermediate lobes in vitro.	gamma-Aminobutyric Acid	65-88	proopiomelanocortin	Homo sapiens	92-101	
3033417-3	1987	High doses of GABA (10(-5) and 10(-4) M) induced a biphasic effect on pars intermedia cells: a brief stimulation followed by a sustained inhibition of alpha-MSH secretion.	gamma-Aminobutyric Acid	14-18	proopiomelanocortin	Homo sapiens	151-160	
3033417-4	1987	Administration of clonazepam (10(-5) M) in the presence of various concentrations of GABA (10(-6) to 10(-4) M) led to a potentiation of both stimulatory and inhibitory phases of alpha-MSH secretion induced by GABA.	gamma-Aminobutyric Acid	85-89	proopiomelanocortin	Homo sapiens	178-187	
3033417-4	1987	Administration of clonazepam (10(-5) M) in the presence of various concentrations of GABA (10(-6) to 10(-4) M) led to a potentiation of both stimulatory and inhibitory phases of alpha-MSH secretion induced by GABA.	gamma-Aminobutyric Acid	209-213	proopiomelanocortin	Homo sapiens	178-187	
3033417-6	1987	Thus our results indicate that endogenous benzodiazepine receptors may modulate the effects of GABA on alpha-MSH secretion.	gamma-Aminobutyric Acid	95-99	proopiomelanocortin	Homo sapiens	103-112