PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29614674-4	2018	In this study, we used ChR2-EGFP driven by doublecortin (DCX) to promote survival and maturation of newborn cells in the dentate gyrus after traumatic brain injury (TBI).	doublecortin	43-55	doublecortin	Mus musculus	57-60	
29614674-11	2018	By administrating verapamil to block L-type calcium channels, we verified that the up-regulation of MAP2, NeuN, Neurog2, NeuroD1 and GluR2 in newborn cells was mediated by ChR2-elicted depolarization.	Verapamil	18-27	microtubule-associated protein 2	Mus musculus	100-104	
29614674-11	2018	By administrating verapamil to block L-type calcium channels, we verified that the up-regulation of MAP2, NeuN, Neurog2, NeuroD1 and GluR2 in newborn cells was mediated by ChR2-elicted depolarization.	Verapamil	18-27	RNA binding protein, fox-1 homolog (C. elegans) 3	Mus musculus	106-110	
29614674-11	2018	By administrating verapamil to block L-type calcium channels, we verified that the up-regulation of MAP2, NeuN, Neurog2, NeuroD1 and GluR2 in newborn cells was mediated by ChR2-elicted depolarization.	Verapamil	18-27	neurogenin 2	Mus musculus	112-119	
29614674-11	2018	By administrating verapamil to block L-type calcium channels, we verified that the up-regulation of MAP2, NeuN, Neurog2, NeuroD1 and GluR2 in newborn cells was mediated by ChR2-elicted depolarization.	Verapamil	18-27	neurogenic differentiation 1	Mus musculus	121-128	
29614674-11	2018	By administrating verapamil to block L-type calcium channels, we verified that the up-regulation of MAP2, NeuN, Neurog2, NeuroD1 and GluR2 in newborn cells was mediated by ChR2-elicted depolarization.	Verapamil	18-27	glutamate receptor, ionotropic, AMPA2 (alpha 2)	Mus musculus	133-138