PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29388696-3	2018	Treatment with caffeic acid phenethyl ester (CAPE), a potent and specific inhibitor that suppresses the translocation of NF-kappaB p65 from the cytoplasm to the nucleus, resulted in decreased levels of RBM3 mRNA and protein and increased incidence of apoptosis despite cells were cultured under hypothermic conditions.	caffeic acid phenethyl ester	15-43	nuclear factor kappa B subunit 1	Homo sapiens	121-130	
29388696-3	2018	Treatment with caffeic acid phenethyl ester (CAPE), a potent and specific inhibitor that suppresses the translocation of NF-kappaB p65 from the cytoplasm to the nucleus, resulted in decreased levels of RBM3 mRNA and protein and increased incidence of apoptosis despite cells were cultured under hypothermic conditions.	caffeic acid phenethyl ester	15-43	RELA proto-oncogene, NF-kB subunit	Homo sapiens	131-134	
29388696-3	2018	Treatment with caffeic acid phenethyl ester (CAPE), a potent and specific inhibitor that suppresses the translocation of NF-kappaB p65 from the cytoplasm to the nucleus, resulted in decreased levels of RBM3 mRNA and protein and increased incidence of apoptosis despite cells were cultured under hypothermic conditions.	caffeic acid phenethyl ester	15-43	RNA binding motif protein 3	Homo sapiens	202-206	
29388696-3	2018	Treatment with caffeic acid phenethyl ester (CAPE), a potent and specific inhibitor that suppresses the translocation of NF-kappaB p65 from the cytoplasm to the nucleus, resulted in decreased levels of RBM3 mRNA and protein and increased incidence of apoptosis despite cells were cultured under hypothermic conditions.	caffeic acid phenethyl ester	45-49	nuclear factor kappa B subunit 1	Homo sapiens	121-130	
29388696-3	2018	Treatment with caffeic acid phenethyl ester (CAPE), a potent and specific inhibitor that suppresses the translocation of NF-kappaB p65 from the cytoplasm to the nucleus, resulted in decreased levels of RBM3 mRNA and protein and increased incidence of apoptosis despite cells were cultured under hypothermic conditions.	caffeic acid phenethyl ester	45-49	RELA proto-oncogene, NF-kB subunit	Homo sapiens	131-134	
29388696-3	2018	Treatment with caffeic acid phenethyl ester (CAPE), a potent and specific inhibitor that suppresses the translocation of NF-kappaB p65 from the cytoplasm to the nucleus, resulted in decreased levels of RBM3 mRNA and protein and increased incidence of apoptosis despite cells were cultured under hypothermic conditions.	caffeic acid phenethyl ester	45-49	RNA binding motif protein 3	Homo sapiens	202-206	
29388696-4	2018	Overexpression of RBM3 abolished the induction of apoptosis in cells treated with CAPE, indicating that NF-kappaB p65-upregulated RBM3 expression is necessary for the suppression of apoptosis.	caffeic acid phenethyl ester	82-86	RNA binding motif protein 3	Homo sapiens	18-22	
29388696-4	2018	Overexpression of RBM3 abolished the induction of apoptosis in cells treated with CAPE, indicating that NF-kappaB p65-upregulated RBM3 expression is necessary for the suppression of apoptosis.	caffeic acid phenethyl ester	82-86	RELA proto-oncogene, NF-kB subunit	Homo sapiens	104-117	
29388696-4	2018	Overexpression of RBM3 abolished the induction of apoptosis in cells treated with CAPE, indicating that NF-kappaB p65-upregulated RBM3 expression is necessary for the suppression of apoptosis.	caffeic acid phenethyl ester	82-86	RNA binding motif protein 3	Homo sapiens	130-134	
29388696-6	2018	Conversely, experiments with cells deficient in RBM3 supported the finding demonstrating that the CAPE-mediated loss of RBM3 induced apoptosis.	caffeic acid phenethyl ester	98-102	RNA binding motif protein 3	Homo sapiens	48-52	
29388696-6	2018	Conversely, experiments with cells deficient in RBM3 supported the finding demonstrating that the CAPE-mediated loss of RBM3 induced apoptosis.	caffeic acid phenethyl ester	98-102	RNA binding motif protein 3	Homo sapiens	120-124