PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29217823-5	2017	LPA administration also strongly blocked the APAP challenge-elicited phosphorylation of JNK, ERK and GSK3beta, which are involved in the pathogenesis of acute liver injury.	lysophosphatidic acid	0-3	mitogen-activated protein kinase 8	Mus musculus	88-91	
29217823-5	2017	LPA administration also strongly blocked the APAP challenge-elicited phosphorylation of JNK, ERK and GSK3beta, which are involved in the pathogenesis of acute liver injury.	lysophosphatidic acid	0-3	mitogen-activated protein kinase 1	Mus musculus	93-96	
29217823-5	2017	LPA administration also strongly blocked the APAP challenge-elicited phosphorylation of JNK, ERK and GSK3beta, which are involved in the pathogenesis of acute liver injury.	lysophosphatidic acid	0-3	glycogen synthase kinase 3 beta	Mus musculus	101-109	
29217823-5	2017	LPA administration also strongly blocked the APAP challenge-elicited phosphorylation of JNK, ERK and GSK3beta, which are involved in the pathogenesis of acute liver injury.	Acetaminophen	45-49	mitogen-activated protein kinase 8	Mus musculus	88-91	
29217823-5	2017	LPA administration also strongly blocked the APAP challenge-elicited phosphorylation of JNK, ERK and GSK3beta, which are involved in the pathogenesis of acute liver injury.	Acetaminophen	45-49	mitogen-activated protein kinase 1	Mus musculus	93-96	
29217823-5	2017	LPA administration also strongly blocked the APAP challenge-elicited phosphorylation of JNK, ERK and GSK3beta, which are involved in the pathogenesis of acute liver injury.	Acetaminophen	45-49	glycogen synthase kinase 3 beta	Mus musculus	101-109	
29217823-6	2017	Furthermore, LPA administration decreased the production of TNF-alpha and IL-1beta in an experimental drug-induced liver injury animal model.	lysophosphatidic acid	13-16	tumor necrosis factor	Mus musculus	60-69	
29217823-6	2017	Furthermore, LPA administration decreased the production of TNF-alpha and IL-1beta in an experimental drug-induced liver injury animal model.	lysophosphatidic acid	13-16	interleukin 1 beta	Mus musculus	74-82	
29217823-9	2017	Collectively, these results indicate that the well-known bioactive lipid LPA can block the pathogenesis of APAP-induced acute liver injury by increasing the glutathione level but decreasing inflammatory cytokines in an LPA1,3,5-independent manner.	lysophosphatidic acid	73-76	lysophosphatidic acid receptor 1	Mus musculus	219-223