PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28743803-0	2017	The glucocorticoid-Angptl4-ceramide axis induces insulin resistance through PP2A and PKCzeta.	Ceramides	27-35	angiopoietin-like 4	Mus musculus	19-26	
28743803-3	2017	Through metabolomic profiling, we revealed that glucocorticoid treatment increased hepatic ceramide concentrations by inducing enzymes in the ceramide synthetic pathway in an Angptl4-dependent manner.	Ceramides	91-99	angiopoietin-like 4	Mus musculus	175-182	
28743803-3	2017	Through metabolomic profiling, we revealed that glucocorticoid treatment increased hepatic ceramide concentrations by inducing enzymes in the ceramide synthetic pathway in an Angptl4-dependent manner.	Ceramides	142-150	angiopoietin-like 4	Mus musculus	175-182	
28743803-4	2017	Angptl4 was also required for glucocorticoids to stimulate the activities of the downstream effectors of ceramide, protein phosphatase 2A (PP2A) and protein kinase Czeta (PKCzeta).	Ceramides	105-113	angiopoietin-like 4	Mus musculus	0-7	
28743803-7	2017	Overall, our study demonstrates the key role of Angptl4 in glucocorticoid-augmented hepatic ceramide production that induces whole-body insulin resistance.	Ceramides	92-100	angiopoietin-like 4	Mus musculus	48-55