PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28445987-2	2017	Here, we identify and study the microRNAs that BRAFV600E regulates through the ERK pathway.By performing small RNA sequencing on A375 melanoma cells and a vemurafenib-resistant clone that was taken as negative control, we discover miR-204 and miR-211 as the miRNAs most induced by vemurafenib.	Vemurafenib	155-166	microRNA 204	Homo sapiens	231-238	
28445987-2	2017	Here, we identify and study the microRNAs that BRAFV600E regulates through the ERK pathway.By performing small RNA sequencing on A375 melanoma cells and a vemurafenib-resistant clone that was taken as negative control, we discover miR-204 and miR-211 as the miRNAs most induced by vemurafenib.	Vemurafenib	155-166	microRNA 211	Homo sapiens	243-250	
28445987-2	2017	Here, we identify and study the microRNAs that BRAFV600E regulates through the ERK pathway.By performing small RNA sequencing on A375 melanoma cells and a vemurafenib-resistant clone that was taken as negative control, we discover miR-204 and miR-211 as the miRNAs most induced by vemurafenib.	Vemurafenib	281-292	microRNA 204	Homo sapiens	231-238	
28445987-2	2017	Here, we identify and study the microRNAs that BRAFV600E regulates through the ERK pathway.By performing small RNA sequencing on A375 melanoma cells and a vemurafenib-resistant clone that was taken as negative control, we discover miR-204 and miR-211 as the miRNAs most induced by vemurafenib.	Vemurafenib	281-292	microRNA 211	Homo sapiens	243-250	
28445987-4	2017	miR-204 is under the control of STAT3 and its expression is induced in amelanotic melanoma cells, where it acts as an effector of vemurafenib"s anti-motility activity by targeting AP1S2.	Vemurafenib	130-141	microRNA 204	Homo sapiens	0-7	
28445987-4	2017	miR-204 is under the control of STAT3 and its expression is induced in amelanotic melanoma cells, where it acts as an effector of vemurafenib"s anti-motility activity by targeting AP1S2.	Vemurafenib	130-141	signal transducer and activator of transcription 3	Homo sapiens	32-37	
28445987-4	2017	miR-204 is under the control of STAT3 and its expression is induced in amelanotic melanoma cells, where it acts as an effector of vemurafenib"s anti-motility activity by targeting AP1S2.	Vemurafenib	130-141	adaptor related protein complex 1 subunit sigma 2	Homo sapiens	180-185	
28445987-6	2017	In doing so, miR-211 serves as an effector of vemurafenib"s pro-pigmentation activity.	Vemurafenib	46-57	microRNA 211	Homo sapiens	13-20	
28445987-7	2017	We also show that such an increase in pigmentation in turn represents an adaptive response that needs to be overcome using appropriate inhibitors in order to increase the efficacy of vemurafenib.In summary, we unveil the distinct and context-dependent activities exerted by miR-204 family members in melanoma cells.	Vemurafenib	183-194	microRNA 204	Homo sapiens	274-281