PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26934053-5	2016	Using mice exposed to THS under conditions that mimic exposure of humans, we show that THS increases cellular oxidative stress by increasing superoxide dismutase (SOD) activity and hydrogen peroxide (H2O2) levels while reducing the activity of antioxidant enzymes catalase and glutathione peroxidase (GPx) that break down H2O2 into H2O and O2.	THYMIDINE-5'-(DITHIO)PHOSPHATE	87-90	catalase	Homo sapiens	264-272	
26934053-9	2016	To determine whether these effects on THS-induced insulin resistance are due to increase in oxidative stress, we treated mice exposed to THS with the antioxidants N-acetyl cysteine (NAC) and alpha-tocopherol (alpha-toc) and showed that the oxidative stress, the molecular damage, and the insulin resistance, were significantly reversed.	THYMIDINE-5'-(DITHIO)PHOSPHATE	38-41	insulin	Homo sapiens	50-57	
26934053-11	2016	In conclusion, THS exposure results in insulin resistance in the form of non-obese type II diabetes (NODII) through oxidative stress.	THYMIDINE-5'-(DITHIO)PHOSPHATE	15-18	insulin	Homo sapiens	39-46