PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26854262-3	2016	This review describes the role of gastrin in the cross-talk between the stomach and the kidney following the ingestion of sodium.	Sodium	122-128	gastrin	Homo sapiens	34-41	
26854262-6	2016	It is proposed that gastrin produced by G-cells via its receptor, cholecystokinin B receptor, interacts with renal D1 -like dopamine receptors to increase renal sodium excretion.	Sodium	161-167	gastrin	Homo sapiens	20-27	
26854262-9	2016	The natriuresis following the ingestion of a certain amount of sodium may be due to an enterokine, gastrin, secreted by G-cells in the stomach and duodenum and released into the circulation.	Sodium	63-69	gastrin	Homo sapiens	99-106	
26854262-12	2016	Gastrin, via its receptor, the cholecystokinin type B receptor (CCKBR), is natriuretic in mammals, including humans, by inhibition of renal sodium transport.	Sodium	140-146	gastrin	Homo sapiens	0-7	
26854262-14	2016	Selective silencing of Gast in the stomach and duodenum impairs the ability to excrete an oral sodium load and also increases blood pressure.	Sodium	95-101	gastrin	Homo sapiens	23-27	
26854262-15	2016	Thus, the gastrorenal axis, mediated by gastrin, can complement pronatriuretic hormones, such as dopamine, to increase sodium excretion after an oral sodium load.	Sodium	119-125	gastrin	Homo sapiens	40-47	
26854262-15	2016	Thus, the gastrorenal axis, mediated by gastrin, can complement pronatriuretic hormones, such as dopamine, to increase sodium excretion after an oral sodium load.	Sodium	150-156	gastrin	Homo sapiens	40-47