PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26803847-5	2016	Nicotine treatment led to dose-dependent increases in the phosphorylation of PERK[Thr981] and eIF2alpha[Ser51], whereas pretreatment with a nicotinic acetylcholine receptor (nAChR) antagonist (mecamylamine hydrochloride) blocked the induction of PERK phosphorylation, verifying the direct involvement of nicotine and nAChR binding.	Nicotine	0-8	cholinergic receptor nicotinic alpha 2 subunit	Rattus norvegicus	140-172	
26803847-5	2016	Nicotine treatment led to dose-dependent increases in the phosphorylation of PERK[Thr981] and eIF2alpha[Ser51], whereas pretreatment with a nicotinic acetylcholine receptor (nAChR) antagonist (mecamylamine hydrochloride) blocked the induction of PERK phosphorylation, verifying the direct involvement of nicotine and nAChR binding.	Nicotine	0-8	cholinergic receptor nicotinic alpha 2 subunit	Rattus norvegicus	174-179	
26803847-5	2016	Nicotine treatment led to dose-dependent increases in the phosphorylation of PERK[Thr981] and eIF2alpha[Ser51], whereas pretreatment with a nicotinic acetylcholine receptor (nAChR) antagonist (mecamylamine hydrochloride) blocked the induction of PERK phosphorylation, verifying the direct involvement of nicotine and nAChR binding.	Nicotine	0-8	cholinergic receptor nicotinic alpha 2 subunit	Rattus norvegicus	317-322	
26803847-5	2016	Nicotine treatment led to dose-dependent increases in the phosphorylation of PERK[Thr981] and eIF2alpha[Ser51], whereas pretreatment with a nicotinic acetylcholine receptor (nAChR) antagonist (mecamylamine hydrochloride) blocked the induction of PERK phosphorylation, verifying the direct involvement of nicotine and nAChR binding.	Nicotine	304-312	cholinergic receptor nicotinic alpha 2 subunit	Rattus norvegicus	174-179