PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26474621-8	2015	In HUVECs, the expressions of CCL-8, Vcam-1, VLA4alpha, OX40 and OX40L were significantly up-regulated by nicotine and P.g-LPS combination compared with nicotine alone, P.g-LPS alone and the untreated control.	Nicotine	106-114	vascular cell adhesion molecule 1	Homo sapiens	37-43	
26474621-10	2015	CONCLUSION: P.g-LPS in combination with nicotine could recruit monocytes to endothelial lesion through up-regulation of CCL-8, and promote adhesion of monocytes to endothelial cells through enhancement of Vcam-1/VLA4alpha and OX40/OX40L interactions, which could be involved in the initiation and development of atherosclerosis.	Nicotine	40-48	vascular cell adhesion molecule 1	Homo sapiens	205-211