PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26259688-5	2015	Therefore, our goal in the present study was to examine the role of microRNA-9 (miR-9) in regulating Nav1.1/Nav1.2 trafficking under CBH generated by bilateral common carotid artery occlusion (2VO).	S-(D-CARBOXYBUTYL)-L-HOMOCYSTEINE	133-136	membrane associated ring-CH-type finger 8	Rattus norvegicus	80-83	
26259688-5	2015	Therefore, our goal in the present study was to examine the role of microRNA-9 (miR-9) in regulating Nav1.1/Nav1.2 trafficking under CBH generated by bilateral common carotid artery occlusion (2VO).	S-(D-CARBOXYBUTYL)-L-HOMOCYSTEINE	133-136	sodium voltage-gated channel alpha subunit 2	Rattus norvegicus	108-114	
26259688-6	2015	RESULTS: The impairment of Nav1.1/Nav1.2 trafficking and decreased expression of Navbeta2 were found in the hippocampi and cortices of rats following CBH generated by bilateral 2VO.	S-(D-CARBOXYBUTYL)-L-HOMOCYSTEINE	150-153	sodium voltage-gated channel alpha subunit 2	Rattus norvegicus	34-40	
26259688-7	2015	MiR-9 was increased in both the hippocampi and cortices of rats following CBH by qRT-PCR.	S-(D-CARBOXYBUTYL)-L-HOMOCYSTEINE	74-77	membrane associated ring-CH-type finger 8	Rattus norvegicus	0-3	
26259688-12	2015	CONCLUSIONS: We conclude that miR-9 may play a key role in regulating the process of Nav1.1/Nav1.2 trafficking via targeting on Navbeta2 protein in 2VO rats at post-transcriptional level, and inhibition of miR-9 may be a potentially valuable approach to prevent Nav1.1/Nav1.2 trafficking disturbance induced by CBH.	S-(D-CARBOXYBUTYL)-L-HOMOCYSTEINE	311-314	membrane associated ring-CH-type finger 8	Rattus norvegicus	30-33	
26259688-12	2015	CONCLUSIONS: We conclude that miR-9 may play a key role in regulating the process of Nav1.1/Nav1.2 trafficking via targeting on Navbeta2 protein in 2VO rats at post-transcriptional level, and inhibition of miR-9 may be a potentially valuable approach to prevent Nav1.1/Nav1.2 trafficking disturbance induced by CBH.	S-(D-CARBOXYBUTYL)-L-HOMOCYSTEINE	311-314	sodium voltage-gated channel alpha subunit 2	Rattus norvegicus	92-98	
26259688-12	2015	CONCLUSIONS: We conclude that miR-9 may play a key role in regulating the process of Nav1.1/Nav1.2 trafficking via targeting on Navbeta2 protein in 2VO rats at post-transcriptional level, and inhibition of miR-9 may be a potentially valuable approach to prevent Nav1.1/Nav1.2 trafficking disturbance induced by CBH.	S-(D-CARBOXYBUTYL)-L-HOMOCYSTEINE	311-314	sodium voltage-gated channel alpha subunit 2	Rattus norvegicus	269-275