PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25794161-0	2015	Genetic and phenotypic variation in UGT2B17, a testosterone-metabolizing enzyme, is associated with BMI in males.	Testosterone	47-59	UDP glucuronosyltransferase family 2 member B17	Homo sapiens	36-43	
25794161-4	2015	In humans, UDP-glucuronosyltransferase 2B17 (UGT2B17) metabolizes testosterone to a metabolite, which is readily excreted in urine.	Testosterone	66-78	UDP glucuronosyltransferase family 2 member B17	Homo sapiens	11-43	
25794161-4	2015	In humans, UDP-glucuronosyltransferase 2B17 (UGT2B17) metabolizes testosterone to a metabolite, which is readily excreted in urine.	Testosterone	66-78	UDP glucuronosyltransferase family 2 member B17	Homo sapiens	45-52	
25794161-7	2015	RESULTS: In Alaska Native people, UGT2B17 deletion was strongly associated with lower BMI in male individuals (P<0.001), but not in female individuals, consistent with testosterone being a male dominant steroid.	Testosterone	171-183	UDP glucuronosyltransferase family 2 member B17	Homo sapiens	34-41	
25794161-12	2015	This is consistent with the hypothesis that reduced UGT2B17-mediated testosterone excretion results in higher testosterone levels.	Testosterone	69-81	UDP glucuronosyltransferase family 2 member B17	Homo sapiens	52-59	
25794161-12	2015	This is consistent with the hypothesis that reduced UGT2B17-mediated testosterone excretion results in higher testosterone levels.	Testosterone	110-122	UDP glucuronosyltransferase family 2 member B17	Homo sapiens	52-59