PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
2565539-3	1989	The mean NAG value corrected for creatinine in the higher cadmium group (2.95 U/g creatinine) is significantly different from the mean low cadmium group value (0.92 U/g creatinine, p = 0.0083).	Cadmium	58-65	N-acetyl-alpha-glucosaminidase	Homo sapiens	9-12	
2565539-3	1989	The mean NAG value corrected for creatinine in the higher cadmium group (2.95 U/g creatinine) is significantly different from the mean low cadmium group value (0.92 U/g creatinine, p = 0.0083).	Creatinine	82-92	N-acetyl-alpha-glucosaminidase	Homo sapiens	9-12	
2565539-3	1989	The mean NAG value corrected for creatinine in the higher cadmium group (2.95 U/g creatinine) is significantly different from the mean low cadmium group value (0.92 U/g creatinine, p = 0.0083).	Creatinine	82-92	N-acetyl-alpha-glucosaminidase	Homo sapiens	9-12	
2565539-5	1989	NAG and AAP also give significant correlations with cadmium levels in urine (NAG: r = 0.51; p = 0.0001; AAP: r = 0.56; p = 0.0001).	Cadmium	52-59	N-acetyl-alpha-glucosaminidase	Homo sapiens	0-3	
2565539-5	1989	NAG and AAP also give significant correlations with cadmium levels in urine (NAG: r = 0.51; p = 0.0001; AAP: r = 0.56; p = 0.0001).	Cadmium	52-59	N-acetyl-alpha-glucosaminidase	Homo sapiens	77-80	
2565539-8	1989	A dose-response relationship was found between NAG and cadmium and between AAP and cadmium.	Cadmium	55-62	N-acetyl-alpha-glucosaminidase	Homo sapiens	47-50	
2565539-8	1989	A dose-response relationship was found between NAG and cadmium and between AAP and cadmium.	Cadmium	83-90	N-acetyl-alpha-glucosaminidase	Homo sapiens	47-50	
2565539-9	1989	The analysis of this relationship gives estimates of a 10% chance of observing an elevated NAG value at a cadmium level of 6.3 micrograms/g creatinine (8.0 micrograms/l) and a 10% chance of observing an elevated AAP at a cadmium level of 5.0 micrograms/g creatinine (3.4 micrograms/l).	Cadmium	106-113	N-acetyl-alpha-glucosaminidase	Homo sapiens	91-94	
2565539-9	1989	The analysis of this relationship gives estimates of a 10% chance of observing an elevated NAG value at a cadmium level of 6.3 micrograms/g creatinine (8.0 micrograms/l) and a 10% chance of observing an elevated AAP at a cadmium level of 5.0 micrograms/g creatinine (3.4 micrograms/l).	Creatinine	140-150	N-acetyl-alpha-glucosaminidase	Homo sapiens	91-94	
2565539-10	1989	These data indicate elevations of NAG and AAP at urine cadmium levels below the level of 10 micrograms/g creatinine recommended as an upper limit by the 1980 World Health Organization Study Group.	Cadmium	55-62	N-acetyl-alpha-glucosaminidase	Homo sapiens	34-37	
2565539-11	1989	Since elevated levels of NAG and AAP in cadmium-exposed workers may reflect chronic renal tubular nephrotoxicity, these findings indicate that cadmium levels below 10 micrograms/g creatinine may be accompanied by subclinical tubular dysfunction, and that WHO guidelines should be interpreted cautiously, particularly with reference to workers who are no longer exposed and may have had higher cadmium body burdens in the past.	Cadmium	40-47	N-acetyl-alpha-glucosaminidase	Homo sapiens	25-28	
2565539-11	1989	Since elevated levels of NAG and AAP in cadmium-exposed workers may reflect chronic renal tubular nephrotoxicity, these findings indicate that cadmium levels below 10 micrograms/g creatinine may be accompanied by subclinical tubular dysfunction, and that WHO guidelines should be interpreted cautiously, particularly with reference to workers who are no longer exposed and may have had higher cadmium body burdens in the past.	Cadmium	143-150	N-acetyl-alpha-glucosaminidase	Homo sapiens	25-28	
2565539-11	1989	Since elevated levels of NAG and AAP in cadmium-exposed workers may reflect chronic renal tubular nephrotoxicity, these findings indicate that cadmium levels below 10 micrograms/g creatinine may be accompanied by subclinical tubular dysfunction, and that WHO guidelines should be interpreted cautiously, particularly with reference to workers who are no longer exposed and may have had higher cadmium body burdens in the past.	Cadmium	143-150	N-acetyl-alpha-glucosaminidase	Homo sapiens	25-28