PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25613382-0	2015	Ketamine treatment partly reverses alterations in brain derived- neurotrophic factor, oxidative stress and energy metabolism parameters induced by an animal model of depression.	Ketamine	0-8	brain-derived neurotrophic factor	Rattus norvegicus	50-84	
25613382-6	2015	In the amygdala, hippocampus and nucleus accumbens (NAc), a reduction in BDNF levels was observed in deprived rats, but the animals treated with ketamine reversed the effects of this animal model only in the amygdala and NAc.	Ketamine	145-153	brain-derived neurotrophic factor	Rattus norvegicus	73-77	
25613382-14	2015	These findings may help to explain that dysfunctions involving BDNF, oxidative stress and energy metabolism within specific brain areas, may be linked with the pathophysiology of depression, and antidepressant effects of ketamine can be positive, at least partially due to the control of these pathways.	Ketamine	221-229	brain-derived neurotrophic factor	Rattus norvegicus	63-67