PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25551612-1	2014	Gaucher disease is a lysosomal storage disease caused by defective activity of acid beta-glucosidase (GCase), which leads to the accumulation of its major substrates, glucosylceramide (GlcCer) and glucosylsphingosine (GlcSph) in many cells.	Glucosylceramides	167-183	glucosidase, beta, acid	Mus musculus	84-100	
25551612-1	2014	Gaucher disease is a lysosomal storage disease caused by defective activity of acid beta-glucosidase (GCase), which leads to the accumulation of its major substrates, glucosylceramide (GlcCer) and glucosylsphingosine (GlcSph) in many cells.	Glucosylceramides	167-183	glucosidase, beta, acid	Mus musculus	102-107	
25551612-1	2014	Gaucher disease is a lysosomal storage disease caused by defective activity of acid beta-glucosidase (GCase), which leads to the accumulation of its major substrates, glucosylceramide (GlcCer) and glucosylsphingosine (GlcSph) in many cells.	Glucosylceramides	185-191	glucosidase, beta, acid	Mus musculus	84-100	
25551612-1	2014	Gaucher disease is a lysosomal storage disease caused by defective activity of acid beta-glucosidase (GCase), which leads to the accumulation of its major substrates, glucosylceramide (GlcCer) and glucosylsphingosine (GlcSph) in many cells.	Glucosylceramides	185-191	glucosidase, beta, acid	Mus musculus	102-107	
25551612-5	2014	GCStg/Gba1 mice showed 2-3 fold increases in tissue GCS activity as well as accelerated GlcCer accumulation and the appearance of lipid-laden CD68 positive macrophages in visceral organs.	Glucosylceramides	88-94	glucosidase, beta, acid	Mus musculus	6-10