PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25233467-6	2014	Results show that nicotine dependence is a mediator of the association between lung adenocarcinoma and gene variations in the regions of CHRNA5/A3/B4 and accounts for approximately 15% of this relationship.	Nicotine	18-26	T cell immune regulator 1, ATPase H+ transporting V0 subunit a3	Homo sapiens	144-149	
25233467-10	2014	Our findings suggest that nicotine dependence plays an important role between genetic variants in the CHRNA5/A3/B4 region, especially CHRNA3, and lung adenocarcinoma.	Nicotine	26-34	T cell immune regulator 1, ATPase H+ transporting V0 subunit a3	Homo sapiens	109-114