PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25179733-5	2015	Functionally, inhibition of MARCKS phosphorylation by enzastaurin or knockdown of the gene by RNAi significantly enhanced the sensitivity of resistant HMCLs and primary MM samples to bortezomib and to other anti-myeloma drugs, providing evidence that MARCKS can modulate drug response.	Bortezomib	183-193	myristoylated alanine rich protein kinase C substrate	Homo sapiens	28-34	
25179733-8	2015	Importantly, MARCKS knockdown in combination with bortezomib treatment overcame bortezomib resistance, significantly inhibited tumor growth and prolonged host survival in a MM xenograft model.	Bortezomib	80-90	myristoylated alanine rich protein kinase C substrate	Homo sapiens	13-19