PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24867956-3	2014	In transformed but not primary murine embryonic fibroblasts, NLRX1 expression mediated resistance to an extrinsic apoptotic signal, whereas conferring susceptibility to intrinsic apoptotic signals, such as glycolysis inhibition, increased cytosolic calcium and endoplasmic reticulum stress.	Calcium	249-256	NLR family member X1	Mus musculus	61-66	
24867956-4	2014	In a murine model of colorectal cancer induced by azoxymethane, NLRX1-/- mice developed fewer tumors than wild type mice.	Azoxymethane	50-62	NLR family member X1	Mus musculus	64-69	
24867956-5	2014	In contrast, in a colitis-associated cancer model combining azoxymethane and dextran sulfate sodium, NLRX1-/- mice developed a more severe pathology likely due to the increased sensitivity to dextran sulfate sodium colitis.	Azoxymethane	60-72	NLR family member X1	Mus musculus	101-106	
24867956-5	2014	In contrast, in a colitis-associated cancer model combining azoxymethane and dextran sulfate sodium, NLRX1-/- mice developed a more severe pathology likely due to the increased sensitivity to dextran sulfate sodium colitis.	Dextran Sulfate	77-99	NLR family member X1	Mus musculus	101-106	
24867956-5	2014	In contrast, in a colitis-associated cancer model combining azoxymethane and dextran sulfate sodium, NLRX1-/- mice developed a more severe pathology likely due to the increased sensitivity to dextran sulfate sodium colitis.	Dextran Sulfate	192-214	NLR family member X1	Mus musculus	101-106