PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23851445-7	2013	Mechanistically, all-trans retinoic acid (ATRA) treatment increased the expression of p14(ARF) in primary human melanocytes and the steady-state levels of p14(ARF) in these cells were shown to be regulated via RARbeta.	Tretinoin	27-40	retinoic acid receptor beta	Homo sapiens	210-217	
23851445-7	2013	Mechanistically, all-trans retinoic acid (ATRA) treatment increased the expression of p14(ARF) in primary human melanocytes and the steady-state levels of p14(ARF) in these cells were shown to be regulated via RARbeta.	Tretinoin	42-46	retinoic acid receptor beta	Homo sapiens	210-217	
23851445-8	2013	Furthermore, the ability of ATRA to induce senescence is reduced in p14(ARF)-depleted melanocytes, and we provide proof-of-concept that ATRA can induce irreversible growth arrest in melanoma cells with an intact RARbeta-p14(ARF) signaling axis, independent of p16(INK4A) and p53 status.	Tretinoin	136-140	retinoic acid receptor beta	Homo sapiens	212-219