PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22613796-4	2012	Whereas Gag-specific CTLs generally were less susceptible to Nef than those targeting other proteins, this was determined by the ability to eliminate infected cells before de novo synthesis of viral proteins, which was also observed for CTLs targeting a Nef epitope.	Glycosaminoglycans	8-11	S100 calcium binding protein B	Homo sapiens	61-64	
22613796-4	2012	Whereas Gag-specific CTLs generally were less susceptible to Nef than those targeting other proteins, this was determined by the ability to eliminate infected cells before de novo synthesis of viral proteins, which was also observed for CTLs targeting a Nef epitope.	Glycosaminoglycans	8-11	S100 calcium binding protein B	Homo sapiens	254-257	
22613796-6	2012	These results suggest that whereas Gag-specific CTLs are more likely to recognize infected cells before Nef-mediated HLA-I down-regulation, this varies depending on the specific epitope and virus inoculum.	Glycosaminoglycans	35-38	S100 calcium binding protein B	Homo sapiens	104-107	
22613796-7	2012	Reduced susceptibility to Nef therefore may contribute to the overall association of Gag-specific CTL responses to better immune control if a sufficient multiplicity of infection is attained in vivo, but this property is not unique to Gag.	Glycosaminoglycans	85-88	S100 calcium binding protein B	Homo sapiens	26-29	
22613796-7	2012	Reduced susceptibility to Nef therefore may contribute to the overall association of Gag-specific CTL responses to better immune control if a sufficient multiplicity of infection is attained in vivo, but this property is not unique to Gag.	Glycosaminoglycans	235-238	S100 calcium binding protein B	Homo sapiens	26-29