PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 22106416-9 2012 Analysis of the link between sAC and apoptosis revealed a sAC and protein kinase A-dependent Bax phosphorylation at Thr(167) and its translocation to mitochondria during SI, which subsequently caused mitochondrial oxygen radical formation followed by cytochrome c release and caspase-9 cleavage during SR. Threonine 116-119 adenylate cyclase 10 Rattus norvegicus 29-32 22106416-9 2012 Analysis of the link between sAC and apoptosis revealed a sAC and protein kinase A-dependent Bax phosphorylation at Thr(167) and its translocation to mitochondria during SI, which subsequently caused mitochondrial oxygen radical formation followed by cytochrome c release and caspase-9 cleavage during SR. Threonine 116-119 adenylate cyclase 10 Rattus norvegicus 58-61 22106416-9 2012 Analysis of the link between sAC and apoptosis revealed a sAC and protein kinase A-dependent Bax phosphorylation at Thr(167) and its translocation to mitochondria during SI, which subsequently caused mitochondrial oxygen radical formation followed by cytochrome c release and caspase-9 cleavage during SR. Threonine 116-119 BCL2 associated X, apoptosis regulator Rattus norvegicus 93-96 22106416-9 2012 Analysis of the link between sAC and apoptosis revealed a sAC and protein kinase A-dependent Bax phosphorylation at Thr(167) and its translocation to mitochondria during SI, which subsequently caused mitochondrial oxygen radical formation followed by cytochrome c release and caspase-9 cleavage during SR. Oxygen 214-220 adenylate cyclase 10 Rattus norvegicus 29-32 22106416-9 2012 Analysis of the link between sAC and apoptosis revealed a sAC and protein kinase A-dependent Bax phosphorylation at Thr(167) and its translocation to mitochondria during SI, which subsequently caused mitochondrial oxygen radical formation followed by cytochrome c release and caspase-9 cleavage during SR. Oxygen 214-220 adenylate cyclase 10 Rattus norvegicus 58-61 22106416-9 2012 Analysis of the link between sAC and apoptosis revealed a sAC and protein kinase A-dependent Bax phosphorylation at Thr(167) and its translocation to mitochondria during SI, which subsequently caused mitochondrial oxygen radical formation followed by cytochrome c release and caspase-9 cleavage during SR. Oxygen 214-220 BCL2 associated X, apoptosis regulator Rattus norvegicus 93-96