PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
21752898-0	2011	Glucose ingestion selectively amplifies ACTH and cortisol secretory-burst mass and enhances their joint synchrony in healthy men.	Glucose	0-7	proopiomelanocortin	Homo sapiens	40-44	
21752898-2	2011	HYPOTHESIS: Glucose ingestion elevates cortisol secretion by 1) augmenting pulsatile ACTH release; and/or 2) enhancing ACTH-cortisol synchrony or dose-responsiveness.	Glucose	12-19	proopiomelanocortin	Homo sapiens	85-89	
21752898-2	2011	HYPOTHESIS: Glucose ingestion elevates cortisol secretion by 1) augmenting pulsatile ACTH release; and/or 2) enhancing ACTH-cortisol synchrony or dose-responsiveness.	Glucose	12-19	proopiomelanocortin	Homo sapiens	119-123	
21752898-7	2011	Glucose ingestion abolished both relationships but amplified pulsatile ACTH (P = 0.009) and cortisol (P = 0.001) secretion.	Glucose	0-7	proopiomelanocortin	Homo sapiens	71-75	
21752898-8	2011	Glucose exposure selectively augmented the mass of ACTH (P < 0.001) and of cortisol (P = 0.004) secreted per burst without altering burst number or basal secretion.	Glucose	0-7	proopiomelanocortin	Homo sapiens	51-55	
21752898-11	2011	According to approximate entropy analysis, glucose input also enhanced the joint synchrony of ACTH-cortisol secretory patterns (P <= 0.001).	Glucose	43-50	proopiomelanocortin	Homo sapiens	94-98	
21752898-13	2011	CONCLUSION: Conjoint augmentation of the mass of ACTH and cortisol secreted per burst and enhancement of ACTH-cortisol synchrony underlie glucose-induced glucocorticoid secretion in healthy men.	Glucose	138-145	proopiomelanocortin	Homo sapiens	49-53	
21752898-13	2011	CONCLUSION: Conjoint augmentation of the mass of ACTH and cortisol secreted per burst and enhancement of ACTH-cortisol synchrony underlie glucose-induced glucocorticoid secretion in healthy men.	Glucose	138-145	proopiomelanocortin	Homo sapiens	105-109