PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
2171946-2	1990	In the absence of verapamil, a rapid increase in radioactivity in phosphatidic acid was observed in platelets stimulated with PAF-acether or thrombin.	Phosphatidic Acids	66-83	prothrombin	Oryctolagus cuniculus	141-149	
2171946-5	1990	When these verapamil-treated platelets were stimulated with PAF-acether or thrombin, there was a rapid, sustained loss of the additional radioactivity induced in the polyphosphoinositides by verapamil.	Verapamil	11-20	prothrombin	Oryctolagus cuniculus	75-83	
2171946-5	1990	When these verapamil-treated platelets were stimulated with PAF-acether or thrombin, there was a rapid, sustained loss of the additional radioactivity induced in the polyphosphoinositides by verapamil.	Phosphatidylinositol Phosphates	166-187	prothrombin	Oryctolagus cuniculus	75-83	
2171946-5	1990	When these verapamil-treated platelets were stimulated with PAF-acether or thrombin, there was a rapid, sustained loss of the additional radioactivity induced in the polyphosphoinositides by verapamil.	Verapamil	191-200	prothrombin	Oryctolagus cuniculus	75-83	
2171946-7	1990	Verapamil may stimulate formation of a separate pool of polyphosphoinositide which is susceptible to agonist-induced phospholipase C, and failure to re-synthesize this polyphosphoinositide could result from inhibition of phosphatidic acid synthesis.	Verapamil	0-9	LOC100009319	Oryctolagus cuniculus	117-132	
2171946-7	1990	Verapamil may stimulate formation of a separate pool of polyphosphoinositide which is susceptible to agonist-induced phospholipase C, and failure to re-synthesize this polyphosphoinositide could result from inhibition of phosphatidic acid synthesis.	Phosphatidylinositol Phosphates	56-76	LOC100009319	Oryctolagus cuniculus	117-132