PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
21368833-12	2011	On the genetic level, FBW7 reconstitution or MCL1 depletion restores sensitivity to ABT-737, establishing MCL1 as a therapeutically relevant bypass survival mechanism that enables FBW7-deficient cells to evade apoptosis.	2,2'-azino-di-(3-ethylbenzothiazoline)-6-sulfonic acid	84-87	MCL1 apoptosis regulator, BCL2 family member	Homo sapiens	106-110