PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
21236267-0	2011	Irbesartan inhibits advanced glycation end product (AGE)-induced up-regulation of vascular cell adhesion molecule-1 (VCAM-1) mRNA levels in glomerular endothelial cells.	Irbesartan	0-10	vascular cell adhesion molecule 1	Homo sapiens	82-115	
21236267-0	2011	Irbesartan inhibits advanced glycation end product (AGE)-induced up-regulation of vascular cell adhesion molecule-1 (VCAM-1) mRNA levels in glomerular endothelial cells.	Irbesartan	0-10	vascular cell adhesion molecule 1	Homo sapiens	117-123	
21236267-4	2011	In this study, we examined whether and how irbesartan, an angiotensin II type 1 receptor blocker (ARB), inhibited the AGE-induced vascular cell adhesion molecule-1 (VCAM-1) gene expression in cultured human glomerular ECs.	Irbesartan	43-53	vascular cell adhesion molecule 1	Homo sapiens	130-163	
21236267-4	2011	In this study, we examined whether and how irbesartan, an angiotensin II type 1 receptor blocker (ARB), inhibited the AGE-induced vascular cell adhesion molecule-1 (VCAM-1) gene expression in cultured human glomerular ECs.	Irbesartan	43-53	vascular cell adhesion molecule 1	Homo sapiens	165-171	
21236267-5	2011	Irbesartan or an anti-oxidant N-acetylcysteine inhibited the AGE-induced increase in reactive oxygen species (ROS) generation and subsequently blocked up-regulation of VCAM-1 mRNA levels in glomerular ECs.	Irbesartan	0-10	vascular cell adhesion molecule 1	Homo sapiens	168-174	
21236267-7	2011	Furthermore, irbesartan completely suppressed up-regulation of VCAM-1 mRNA levels in AGE plus angiotensin II-exposed glomerular ECs.	Irbesartan	13-23	vascular cell adhesion molecule 1	Homo sapiens	63-69