PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20151649-0	2010	Gallic acid induces apoptosis of lung fibroblasts via a reactive oxygen species-dependent ataxia telangiectasia mutated-p53 activation pathway.	Gallic Acid	0-11	ataxia telangiectasia mutated	Mus musculus	90-119	
20151649-9	2010	Mechanistic studies showed that gallic acid induces early phosphorylation of p53(Ser18) and histone 2AX(Ser139) (H2AX) via ataxia telangiectasia mutated (ATM) activation in response to ROS-provoked DNA damage.	Gallic Acid	32-43	ataxia telangiectasia mutated	Mus musculus	123-152	
20151649-9	2010	Mechanistic studies showed that gallic acid induces early phosphorylation of p53(Ser18) and histone 2AX(Ser139) (H2AX) via ataxia telangiectasia mutated (ATM) activation in response to ROS-provoked DNA damage.	Gallic Acid	32-43	ataxia telangiectasia mutated	Mus musculus	154-157	
20151649-10	2010	When mouse lung fibroblasts were treated with caffeine, an ATM kinase inhibitor, the levels of p53, phosphorylated p53(Ser18), and cell death induced by gallic acid were significantly attenuated.	Gallic Acid	153-164	ataxia telangiectasia mutated	Mus musculus	59-62	
20151649-11	2010	Additionally, pretreatment with antioxidants drastically inhibited the gallic acid-induced 8-hydroxy-2"-deoxyguanosine (8-OH-dG) formation and phosphorylation of p53(Ser18) and ATM(Ser1981), as well as apoptosis.	Gallic Acid	71-82	ataxia telangiectasia mutated	Mus musculus	177-180	
20151649-12	2010	Our results provide the first evidence of the activation of ROS-dependent ATM/p53 signaling as a critical mechanism of gallic acid-induced cell death in primary cultured mouse lung fibroblasts.	Gallic Acid	119-130	ataxia telangiectasia mutated	Mus musculus	74-77