PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20055451-0	2010	2,3,7,8-Tetrachlorodibenzo-p-dioxin upregulates FoxQ1b in zebrafish jaw primordium.	Polychlorinated Dibenzodioxins	0-35	forkhead box Q1b	Danio rerio	48-54	
20055451-1	2010	Vertebrate jaw development can be disrupted by exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-a potent activator of the aryl hydrocarbon receptor (AHR) transcription factor required for transducing the toxic effects of TCDD.	Polychlorinated Dibenzodioxins	59-94	aryl hydrocarbon receptor 1a	Danio rerio	128-153	
20055451-1	2010	Vertebrate jaw development can be disrupted by exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-a potent activator of the aryl hydrocarbon receptor (AHR) transcription factor required for transducing the toxic effects of TCDD.	Polychlorinated Dibenzodioxins	59-94	aryl hydrocarbon receptor 1a	Danio rerio	155-158	
20055451-1	2010	Vertebrate jaw development can be disrupted by exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-a potent activator of the aryl hydrocarbon receptor (AHR) transcription factor required for transducing the toxic effects of TCDD.	Polychlorinated Dibenzodioxins	96-100	aryl hydrocarbon receptor 1a	Danio rerio	128-153	
20055451-1	2010	Vertebrate jaw development can be disrupted by exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-a potent activator of the aryl hydrocarbon receptor (AHR) transcription factor required for transducing the toxic effects of TCDD.	Polychlorinated Dibenzodioxins	96-100	aryl hydrocarbon receptor 1a	Danio rerio	155-158	
20055451-1	2010	Vertebrate jaw development can be disrupted by exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-a potent activator of the aryl hydrocarbon receptor (AHR) transcription factor required for transducing the toxic effects of TCDD.	Polychlorinated Dibenzodioxins	227-231	aryl hydrocarbon receptor 1a	Danio rerio	128-153	
20055451-1	2010	Vertebrate jaw development can be disrupted by exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-a potent activator of the aryl hydrocarbon receptor (AHR) transcription factor required for transducing the toxic effects of TCDD.	Polychlorinated Dibenzodioxins	227-231	aryl hydrocarbon receptor 1a	Danio rerio	155-158	
20055451-3	2010	Our results uncovered a novel target of TCDD-activated Ahr belonging to the evolutionarily conserved family of forkhead box transcription factors.	Polychlorinated Dibenzodioxins	40-44	aryl hydrocarbon receptor 1a	Danio rerio	55-58	
20055451-4	2010	Quantitative real-time polymerase chain reaction analysis demonstrated that FoxQ1b was upregulated by TCDD 7- and 10-fold at 24 and 48 h postfertilization (hpf), respectively.	Polychlorinated Dibenzodioxins	102-106	forkhead box Q1b	Danio rerio	76-82	
20055451-5	2010	The rate of TCDD-induced FoxQ1b expression was more rapid than that of Cyp1a, a known direct target of TCDD-activated Ahr.	Polychlorinated Dibenzodioxins	12-16	forkhead box Q1b	Danio rerio	25-31	
20055451-5	2010	The rate of TCDD-induced FoxQ1b expression was more rapid than that of Cyp1a, a known direct target of TCDD-activated Ahr.	Polychlorinated Dibenzodioxins	12-16	aryl hydrocarbon receptor 1a	Danio rerio	118-121	
20055451-5	2010	The rate of TCDD-induced FoxQ1b expression was more rapid than that of Cyp1a, a known direct target of TCDD-activated Ahr.	Polychlorinated Dibenzodioxins	103-107	cytochrome P450, family 1, subfamily A	Danio rerio	71-76	
20055451-5	2010	The rate of TCDD-induced FoxQ1b expression was more rapid than that of Cyp1a, a known direct target of TCDD-activated Ahr.	Polychlorinated Dibenzodioxins	103-107	aryl hydrocarbon receptor 1a	Danio rerio	118-121	
20055451-6	2010	TCDD-mediated induction of FoxQ1b was suppressed in the presence of an Ahr antagonist, alpha-naphthoflavone, as well as following knockdown of Ahr2 expression using an Ahr2-specific morpholino antisense oligonucleotide.	Polychlorinated Dibenzodioxins	0-4	forkhead box Q1b	Danio rerio	27-33	
20055451-6	2010	TCDD-mediated induction of FoxQ1b was suppressed in the presence of an Ahr antagonist, alpha-naphthoflavone, as well as following knockdown of Ahr2 expression using an Ahr2-specific morpholino antisense oligonucleotide.	Polychlorinated Dibenzodioxins	0-4	aryl hydrocarbon receptor 1a	Danio rerio	71-74	
20055451-6	2010	TCDD-mediated induction of FoxQ1b was suppressed in the presence of an Ahr antagonist, alpha-naphthoflavone, as well as following knockdown of Ahr2 expression using an Ahr2-specific morpholino antisense oligonucleotide.	Polychlorinated Dibenzodioxins	0-4	aryl hydrocarbon receptor 2	Danio rerio	143-147	
20055451-6	2010	TCDD-mediated induction of FoxQ1b was suppressed in the presence of an Ahr antagonist, alpha-naphthoflavone, as well as following knockdown of Ahr2 expression using an Ahr2-specific morpholino antisense oligonucleotide.	Polychlorinated Dibenzodioxins	0-4	aryl hydrocarbon receptor 2	Danio rerio	168-172	
20055451-6	2010	TCDD-mediated induction of FoxQ1b was suppressed in the presence of an Ahr antagonist, alpha-naphthoflavone, as well as following knockdown of Ahr2 expression using an Ahr2-specific morpholino antisense oligonucleotide.	alpha-naphthoflavone	87-107	forkhead box Q1b	Danio rerio	27-33	
20055451-6	2010	TCDD-mediated induction of FoxQ1b was suppressed in the presence of an Ahr antagonist, alpha-naphthoflavone, as well as following knockdown of Ahr2 expression using an Ahr2-specific morpholino antisense oligonucleotide.	alpha-naphthoflavone	87-107	aryl hydrocarbon receptor 1a	Danio rerio	71-74	
20055451-6	2010	TCDD-mediated induction of FoxQ1b was suppressed in the presence of an Ahr antagonist, alpha-naphthoflavone, as well as following knockdown of Ahr2 expression using an Ahr2-specific morpholino antisense oligonucleotide.	Morpholinos	182-192	forkhead box Q1b	Danio rerio	27-33	
20055451-6	2010	TCDD-mediated induction of FoxQ1b was suppressed in the presence of an Ahr antagonist, alpha-naphthoflavone, as well as following knockdown of Ahr2 expression using an Ahr2-specific morpholino antisense oligonucleotide.	Morpholinos	182-192	aryl hydrocarbon receptor 2	Danio rerio	143-147	
20055451-6	2010	TCDD-mediated induction of FoxQ1b was suppressed in the presence of an Ahr antagonist, alpha-naphthoflavone, as well as following knockdown of Ahr2 expression using an Ahr2-specific morpholino antisense oligonucleotide.	Morpholinos	182-192	aryl hydrocarbon receptor 2	Danio rerio	168-172	
20055451-6	2010	TCDD-mediated induction of FoxQ1b was suppressed in the presence of an Ahr antagonist, alpha-naphthoflavone, as well as following knockdown of Ahr2 expression using an Ahr2-specific morpholino antisense oligonucleotide.	Oligonucleotides	203-218	forkhead box Q1b	Danio rerio	27-33	
20055451-6	2010	TCDD-mediated induction of FoxQ1b was suppressed in the presence of an Ahr antagonist, alpha-naphthoflavone, as well as following knockdown of Ahr2 expression using an Ahr2-specific morpholino antisense oligonucleotide.	Oligonucleotides	203-218	aryl hydrocarbon receptor 2	Danio rerio	143-147	
20055451-6	2010	TCDD-mediated induction of FoxQ1b was suppressed in the presence of an Ahr antagonist, alpha-naphthoflavone, as well as following knockdown of Ahr2 expression using an Ahr2-specific morpholino antisense oligonucleotide.	Oligonucleotides	203-218	aryl hydrocarbon receptor 2	Danio rerio	168-172	
20055451-7	2010	In situ hybridization analysis of FoxQ1b expression at 48 hpf demonstrated that FoxQ1b is specifically expressed in the jaw primordium where it discretely outlines a developing jaw structure known as Meckel"s cartilage--a conserved structure in all jawed vertebrates that develops abnormally in the presence of TCDD.	Polychlorinated Dibenzodioxins	311-315	forkhead box Q1b	Danio rerio	80-86	
20055451-8	2010	These results identify a novel target of TCDD-activated Ahr and suggest that FoxQ1b may play a role in craniofacial abnormalities induced by developmental exposure to TCDD.	Polychlorinated Dibenzodioxins	41-45	aryl hydrocarbon receptor 1a	Danio rerio	56-59	
20055451-8	2010	These results identify a novel target of TCDD-activated Ahr and suggest that FoxQ1b may play a role in craniofacial abnormalities induced by developmental exposure to TCDD.	Polychlorinated Dibenzodioxins	41-45	forkhead box Q1b	Danio rerio	77-83	
20055451-8	2010	These results identify a novel target of TCDD-activated Ahr and suggest that FoxQ1b may play a role in craniofacial abnormalities induced by developmental exposure to TCDD.	Polychlorinated Dibenzodioxins	167-171	aryl hydrocarbon receptor 1a	Danio rerio	56-59	
20055451-8	2010	These results identify a novel target of TCDD-activated Ahr and suggest that FoxQ1b may play a role in craniofacial abnormalities induced by developmental exposure to TCDD.	Polychlorinated Dibenzodioxins	167-171	forkhead box Q1b	Danio rerio	77-83