PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20025056-0	2010	Curcumin protects against hepatic and renal injuries mediated by inducible nitric oxide synthase during selenium-induced toxicity in Wistar rats.	Curcumin	0-8	nitric oxide synthase 2	Rattus norvegicus	65-96	
20025056-8	2010	To understand the probable mechanism of action of curcumin, we analyzed inducible nitric oxide synthase (iNOS) expression by immunohistochemistry, and the results showed an increased iNOS expression in selenium-alone induced liver and kidney.	Curcumin	50-58	nitric oxide synthase 2	Rattus norvegicus	72-103	
20025056-8	2010	To understand the probable mechanism of action of curcumin, we analyzed inducible nitric oxide synthase (iNOS) expression by immunohistochemistry, and the results showed an increased iNOS expression in selenium-alone induced liver and kidney.	Curcumin	50-58	nitric oxide synthase 2	Rattus norvegicus	183-187	
20025056-9	2010	Such high iNOS levels were inhibited in liver and kidney of rats pretreated with curcumin and then with selenium 24 h later.	Curcumin	81-89	nitric oxide synthase 2	Rattus norvegicus	10-14	
20025056-10	2010	Based on the histological results, it can be concluded that curcumin functions as a protective agent against selenium-induced toxicity in liver as well as kidney, and this action is probably by the regulatory role of curcumin on iNOS expression.	Curcumin	60-68	nitric oxide synthase 2	Rattus norvegicus	229-233	
20025056-10	2010	Based on the histological results, it can be concluded that curcumin functions as a protective agent against selenium-induced toxicity in liver as well as kidney, and this action is probably by the regulatory role of curcumin on iNOS expression.	Curcumin	217-225	nitric oxide synthase 2	Rattus norvegicus	229-233