PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 19340428-0 2009 IGHV gene mutational status and LPL/ADAM29 gene expression as clinical outcome predictors in CLL patients in remission following treatment with oral fludarabine plus cyclophosphamide. Cyclophosphamide 166-182 immunoglobulin heavy variable 3-69-1 (pseudogene) Homo sapiens 0-4 19340428-0 2009 IGHV gene mutational status and LPL/ADAM29 gene expression as clinical outcome predictors in CLL patients in remission following treatment with oral fludarabine plus cyclophosphamide. Cyclophosphamide 166-182 lipoprotein lipase Homo sapiens 32-35 19340428-10 2009 Our results support the use of LPL and ADAM29 gene expression associated to IGHV mutational status for predicting the clinical outcome of patients treated by oral fludarabine + cyclophosphamide and could be considered for treatment strategies. fludarabine 163-174 lipoprotein lipase Homo sapiens 31-34 19340428-10 2009 Our results support the use of LPL and ADAM29 gene expression associated to IGHV mutational status for predicting the clinical outcome of patients treated by oral fludarabine + cyclophosphamide and could be considered for treatment strategies. fludarabine 163-174 ADAM metallopeptidase domain 29 Homo sapiens 39-45 19340428-10 2009 Our results support the use of LPL and ADAM29 gene expression associated to IGHV mutational status for predicting the clinical outcome of patients treated by oral fludarabine + cyclophosphamide and could be considered for treatment strategies. fludarabine 163-174 immunoglobulin heavy variable 3-69-1 (pseudogene) Homo sapiens 76-80 19340428-10 2009 Our results support the use of LPL and ADAM29 gene expression associated to IGHV mutational status for predicting the clinical outcome of patients treated by oral fludarabine + cyclophosphamide and could be considered for treatment strategies. Cyclophosphamide 177-193 lipoprotein lipase Homo sapiens 31-34 19340428-10 2009 Our results support the use of LPL and ADAM29 gene expression associated to IGHV mutational status for predicting the clinical outcome of patients treated by oral fludarabine + cyclophosphamide and could be considered for treatment strategies. Cyclophosphamide 177-193 ADAM metallopeptidase domain 29 Homo sapiens 39-45 19340428-10 2009 Our results support the use of LPL and ADAM29 gene expression associated to IGHV mutational status for predicting the clinical outcome of patients treated by oral fludarabine + cyclophosphamide and could be considered for treatment strategies. Cyclophosphamide 177-193 immunoglobulin heavy variable 3-69-1 (pseudogene) Homo sapiens 76-80