PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19188434-0	2009	High glucose suppresses epidermal growth factor receptor/phosphatidylinositol 3-kinase/Akt signaling pathway and attenuates corneal epithelial wound healing.	Glucose	5-12	epidermal growth factor receptor	Homo sapiens	24-56	
19188434-2	2009	This study investigated the effects of high glucose on epidermal growth factor receptor (EGFR) signaling and on epithelial wound healing in the cornea.	Glucose	44-51	epidermal growth factor receptor	Homo sapiens	55-87	
19188434-6	2009	RESULTS: High glucose impaired ex vivo epithelial wound healing and disturbed cell responses and EGFR signaling to wounding.	Glucose	14-21	epidermal growth factor receptor	Homo sapiens	97-101	
19188434-11	2009	CONCLUSIONS: High glucose, likely through ROS, impairs the EGFR-phosphatidylinositol 3-kinase/Akt pathway, resulting in delayed corneal epithelial wound healing.	Glucose	18-25	epidermal growth factor receptor	Homo sapiens	59-63