PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19176463-0	2009	The role of STAT-3 in the induction of apoptosis in pancreatic cancer cells by benzyl isothiocyanate.	benzyl isothiocyanate	79-100	signal transducer and activator of transcription 3	Homo sapiens	12-18	
19176463-4	2009	The effects of BITC treatment on tumor growth, apoptosis, and STAT-3 protein expression in vivo were studied in xenografts of BxPC-3 pancreatic tumor cells in athymic nude mice.	benzyl isothiocyanate	15-19	signal transducer and activator of transcription 3	Homo sapiens	62-68	
19176463-8	2009	Overexpression of STAT-3 in BxPC-3 cells inhibited BITC-induced apoptosis and restored STAT-3 activity.	benzyl isothiocyanate	51-55	signal transducer and activator of transcription 3	Homo sapiens	18-24	
19176463-10	2009	CONCLUSION: BITC induces apoptosis in some types of pancreatic cancer cells by inhibiting the STAT-3 signaling pathway.	benzyl isothiocyanate	12-16	signal transducer and activator of transcription 3	Homo sapiens	94-100