PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
18751369-0	2008	RET activation inhibits doxorubicin-induced apoptosis in SK-N-MC cells.	sk-n-mc	57-64	ret proto-oncogene	Homo sapiens	0-3	
18751369-2	2008	MATERIALS AND METHODS: Each RET isoform was separately expressed in SK-N-MC cells (neural crest-derived tumor) and the impact of RET activation on doxorubicin-induced apoptosis was examined.	sk-n-mc	68-75	ret proto-oncogene	Homo sapiens	28-31	
18751369-3	2008	RESULTS: The activation of RET9 and RET51 in the SK-N-MC cells significantly reduced the doxorubicin-induced apoptosis by 50%, compared to untreated cells.	sk-n-mc	49-56	ret proto-oncogene	Homo sapiens	36-41	
18751369-6	2008	CONCLUSION: In SK-N-MC cells, downstream activation of MAP kinase, by both RET9 and RET51, appears to mediate the majority of RET-dependent resistance to chemotherapeutically induced apoptosis.	sk-n-mc	15-22	ret proto-oncogene	Homo sapiens	84-89	
18751369-6	2008	CONCLUSION: In SK-N-MC cells, downstream activation of MAP kinase, by both RET9 and RET51, appears to mediate the majority of RET-dependent resistance to chemotherapeutically induced apoptosis.	sk-n-mc	15-22	ret proto-oncogene	Homo sapiens	75-78