PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
17633456-8	2007	Sodium selenite also downregulated Bcl-xl and activated Bax and Bid at protein level.	Sodium Selenite	0-15	BH3 interacting domain death agonist	Homo sapiens	64-67	
17633456-9	2007	Pretreatment with antioxidant MnTmPy almost fully abrogated the proapoptotic effect of sodium selenite prevented the cleavage of Bid protein and in turn the mitochondrail transmembrane potential loss.	Sodium Selenite	87-102	BH3 interacting domain death agonist	Homo sapiens	129-132	
17633456-11	2007	CONCLUSIONS: Sodium selenite may induce apoptosis by inducing ROS production in NB4 cells, which leads to the downregulation of Bcl-xl, upregulation of Bax, and cleavage and activation of Bid.	Sodium Selenite	13-28	BH3 interacting domain death agonist	Homo sapiens	188-191