PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16049007-10	2005	Furthermore, H(2)O(2) treatment not only induced GST expression but also increased intracellular maspin/GST interaction, which was inversely correlated with the level of ROS generation.	Reactive Oxygen Species	170-173	glutathione S-transferase kappa 1	Homo sapiens	104-107	
16049007-12	2005	Furthermore, the maspin effect on ROS generation was completely abolished by a GST inhibitor, indicating an essential role of GST in maspin-mediated cellular response to oxidative stress.	Reactive Oxygen Species	34-37	glutathione S-transferase kappa 1	Homo sapiens	79-82	
16049007-12	2005	Furthermore, the maspin effect on ROS generation was completely abolished by a GST inhibitor, indicating an essential role of GST in maspin-mediated cellular response to oxidative stress.	Reactive Oxygen Species	34-37	glutathione S-transferase kappa 1	Homo sapiens	126-129	
16049007-14	2005	Together, our data suggest a new mechanism by which maspin, through its direct interaction with GST, may inhibit oxidative stress-induced ROS generation and vascular endothelial growth factor A induction, thus preventing further adverse effects on tumor genetics and stromal reactivity.	Reactive Oxygen Species	138-141	glutathione S-transferase kappa 1	Homo sapiens	96-99