PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15820254-9	2005	CONCLUSIONS: The data demonstrate that the pathway of NF-kappaB activation involving serine phosphorylation of IkappaBalpha is not the primary mechanism for induction of liver inflammation after ischemia/reperfusion and suggest that alternative pathways, such as tyrosine phosphorylation of IkappaBalpha, may be essential for the postischemic response in liver.	Tyrosine	263-271	nuclear factor of kappa light polypeptide gene enhancer in B cells inhibitor, alpha	Mus musculus	111-123