PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15774489-0	2005	Transformation of immortalized colorectal crypt cells by microcystin involving constitutive activation of Akt and MAPK cascade.	microcystin	57-68	AKT serine/threonine kinase 1	Homo sapiens	106-109	
15774489-10	2005	Inhibition of Akt, p38 and JNK activation led to a reduced proliferation of microcystin-transformed cells.	microcystin	76-87	AKT serine/threonine kinase 1	Homo sapiens	14-17	
15774489-10	2005	Inhibition of Akt, p38 and JNK activation led to a reduced proliferation of microcystin-transformed cells.	microcystin	76-87	mitogen-activated protein kinase 14	Homo sapiens	19-22	
15774489-10	2005	Inhibition of Akt, p38 and JNK activation led to a reduced proliferation of microcystin-transformed cells.	microcystin	76-87	mitogen-activated protein kinase 8	Homo sapiens	27-30	
15774489-11	2005	This implies that the constitutive activation of Akt and the p38, JNK of MAPK pathways in microcystin-transformed cells may be the mechanism by which this important external factor acts in the carcinogenesis of CRC.	microcystin	90-101	AKT serine/threonine kinase 1	Homo sapiens	49-52	
15774489-11	2005	This implies that the constitutive activation of Akt and the p38, JNK of MAPK pathways in microcystin-transformed cells may be the mechanism by which this important external factor acts in the carcinogenesis of CRC.	microcystin	90-101	mitogen-activated protein kinase 14	Homo sapiens	61-64	
15774489-11	2005	This implies that the constitutive activation of Akt and the p38, JNK of MAPK pathways in microcystin-transformed cells may be the mechanism by which this important external factor acts in the carcinogenesis of CRC.	microcystin	90-101	mitogen-activated protein kinase 8	Homo sapiens	66-69