PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15454728-2	2004	CYP3A4 expression is highly variable and is induced by numerous compounds of exogenous and endogenous origin, including elevated concentrations of secondary bile acids via the pregnane X receptor (PXR).	Bile Acids and Salts	157-167	cytochrome P450 family 3 subfamily A member 4	Homo sapiens	0-6	
15454728-2	2004	CYP3A4 expression is highly variable and is induced by numerous compounds of exogenous and endogenous origin, including elevated concentrations of secondary bile acids via the pregnane X receptor (PXR).	Bile Acids and Salts	157-167	nuclear receptor subfamily 1, group I, member 2	Mus musculus	176-195	
15454728-2	2004	CYP3A4 expression is highly variable and is induced by numerous compounds of exogenous and endogenous origin, including elevated concentrations of secondary bile acids via the pregnane X receptor (PXR).	Bile Acids and Salts	157-167	nuclear receptor subfamily 1, group I, member 2	Mus musculus	197-200	
15454728-3	2004	We show that physiological concentrations of the primary bile acid chenodeoxycholic acid regulate the expression of CYP3A4 via the bile acid receptor FXR.	Bile Acids and Salts	57-66	cytochrome P450 family 3 subfamily A member 4	Homo sapiens	116-122	
15454728-3	2004	We show that physiological concentrations of the primary bile acid chenodeoxycholic acid regulate the expression of CYP3A4 via the bile acid receptor FXR.	Bile Acids and Salts	57-66	nuclear receptor subfamily 1, group H, member 4	Mus musculus	150-153	
15454728-3	2004	We show that physiological concentrations of the primary bile acid chenodeoxycholic acid regulate the expression of CYP3A4 via the bile acid receptor FXR.	Chenodeoxycholic Acid	67-88	cytochrome P450 family 3 subfamily A member 4	Homo sapiens	116-122	
15454728-3	2004	We show that physiological concentrations of the primary bile acid chenodeoxycholic acid regulate the expression of CYP3A4 via the bile acid receptor FXR.	Chenodeoxycholic Acid	67-88	nuclear receptor subfamily 1, group H, member 4	Mus musculus	150-153	
15454728-7	2004	Thus, whereas elevated concentrations of precursors of bile acids and secondary bile acids induce CYP3A via PXR, primary bile acids can modulate the expression of CYP3A via FXR.	Bile Acids and Salts	55-65	cytochrome P450, family 3, subfamily a, polypeptide 11	Mus musculus	98-103	
15454728-7	2004	Thus, whereas elevated concentrations of precursors of bile acids and secondary bile acids induce CYP3A via PXR, primary bile acids can modulate the expression of CYP3A via FXR.	Bile Acids and Salts	55-65	nuclear receptor subfamily 1, group I, member 2	Mus musculus	108-111	
15454728-7	2004	Thus, whereas elevated concentrations of precursors of bile acids and secondary bile acids induce CYP3A via PXR, primary bile acids can modulate the expression of CYP3A via FXR.	Bile Acids and Salts	80-90	cytochrome P450, family 3, subfamily a, polypeptide 11	Mus musculus	98-103	
15454728-7	2004	Thus, whereas elevated concentrations of precursors of bile acids and secondary bile acids induce CYP3A via PXR, primary bile acids can modulate the expression of CYP3A via FXR.	Bile Acids and Salts	80-90	nuclear receptor subfamily 1, group I, member 2	Mus musculus	108-111	
15454728-7	2004	Thus, whereas elevated concentrations of precursors of bile acids and secondary bile acids induce CYP3A via PXR, primary bile acids can modulate the expression of CYP3A via FXR.	Bile Acids and Salts	80-90	cytochrome P450, family 3, subfamily a, polypeptide 11	Mus musculus	98-103	
15454728-7	2004	Thus, whereas elevated concentrations of precursors of bile acids and secondary bile acids induce CYP3A via PXR, primary bile acids can modulate the expression of CYP3A via FXR.	Bile Acids and Salts	80-90	nuclear receptor subfamily 1, group I, member 2	Mus musculus	108-111