PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15371330-5	2004	H2S treatment increased the activities of ERK and p38 mitogen-activated protein kinase (MAPK), but not c-Jun N-terminal kinase activity.	Hydrogen Sulfide	0-3	mitogen-activated protein kinase 1	Homo sapiens	42-45	
15371330-5	2004	H2S treatment increased the activities of ERK and p38 mitogen-activated protein kinase (MAPK), but not c-Jun N-terminal kinase activity.	Hydrogen Sulfide	0-3	mitogen-activated protein kinase 1	Homo sapiens	88-92	
15371330-6	2004	Suppression of extracellular signal-regulated kinase (ERK) activity, but not of p38 activity, inhibited the H2S-induced apoptosis of HASMCs.	Hydrogen Sulfide	108-111	mitogen-activated protein kinase 1	Homo sapiens	15-52	
15371330-6	2004	Suppression of extracellular signal-regulated kinase (ERK) activity, but not of p38 activity, inhibited the H2S-induced apoptosis of HASMCs.	Hydrogen Sulfide	108-111	mitogen-activated protein kinase 1	Homo sapiens	54-57	
15371330-7	2004	The activation of ERK by H2S in HASMCs was accompanied by increased caspase-3 activity.	Hydrogen Sulfide	25-28	mitogen-activated protein kinase 1	Homo sapiens	18-21	
15371330-11	2004	Of the three investigated MAPKs, only ERK played an active role in mediating H2S-induced apoptosis of HASMCs by activating caspase-3.	Hydrogen Sulfide	77-80	mitogen-activated protein kinase 1	Homo sapiens	38-41