PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15329596-0	2004	Sevoflurane inhibits phorbol-myristate-acetate-induced activator protein-1 activation in human T lymphocytes in vitro: potential role of the p38-stress kinase pathway.	Sevoflurane	0-11	JunB proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	55-74	
15329596-7	2004	RESULTS: Sevoflurane inhibited activation of the transcription factor AP-1.	Sevoflurane	9-20	JunB proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	70-74	
15329596-9	2004	Sevoflurane-mediated suppression of AP-1 could be observed in primary CD3 lymphocytes from healthy volunteers, was time-dependent and concentration-dependent, and occurred at concentrations that are clinically achieved.	Sevoflurane	0-11	JunB proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	36-40	
15329596-12	2004	CONCLUSION: The data demonstrate that sevoflurane is a specific inhibitor of AP-1 and may thus provide a molecular mechanism for the antiinflammatory effects associated with sevoflurane administration.	Sevoflurane	38-49	JunB proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	77-81	
15329596-12	2004	CONCLUSION: The data demonstrate that sevoflurane is a specific inhibitor of AP-1 and may thus provide a molecular mechanism for the antiinflammatory effects associated with sevoflurane administration.	Sevoflurane	174-185	JunB proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	77-81